Whether your focus is on losing weight, healthy eating or just looking and feeling better, the chances are you know you should cut back on sugar.
But that’s easier said than done. Few of us are immune from a ‘deep, deep and ancient craving’ for sweet foods. While fruit and honey provided our pre-historic ancestors with essential fuel for survival, today, our sugar intake is blamed for a worldwide epidemic of obesity and heart disease!
But just how did natural human enjoyment of sweet foods become a health risk? Let’s take a closer look at sugar, what eating too much sugar does to the human body, and tips to reduce our intake.
Sugar, or more accurately sugars, are simple carbohydrates that our bodies need to function. Sugar is the fuel for the cells of the human body, brain and nervous system. The excess is stored in our body as fat.
Sugar molecules are chemically the same, whether found in fruit, or added in foods or beverages. All sugars break down in the small intestine and enter the blood as glucose. In this case, why does it matter whether you choose to get your energy from a banana, or a regular Oreo McFlurry?
One school of thought says it doesn’t matter. Firstly, this ignores the sheer quantity of sugar ingested in one go in a dessert like a McFlurry; a whopping 63 g of pure glucose, roughly double an adult’s daily recommended limit!
Secondly, without fibre and other nutrients to slow it down, the sugar in the McFlurry enters your bloodstream fast. This causes glucose levels to rise sharply, then plummet as the hormone insulin from the pancreas signals the cells of the body to open up and take in the glucose. Spikes like this in glucose levels in the blood can lead to mood swings, fatigue, and cravings for sweet foods.
Thirdly, the intense sweetness of added sugars literally lights up the pleasure/reward spots in our brain. As a result we tend to over-eat. As these foods provide largely empty calories of low nutritional value, we feel hungry again soon after eating.
On the other hand, a banana raises glucose levels slowly. Firstly, the various sugars take time to break down into glucose. Then fibre slows the rate glucose is absorbed.
Unlike the McFlurry, a banana is both rich in nutrients, and filling. What’s more, they are difficult to over-eat. Very few of us find eating bananas so enjoyable that we could eat 4 or 5 in one sitting for a McFlurry-style high (always exceptions- you know who you are!).
So while sugar molecules may all look the same under a microscope, in our bodies, the effect is quite different.
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Sugar is so familiar and common that it is difficult to imagine life without it. Yet it is only in the last 250 years that it has become widely available; first as sucrose in table sugar, and later, as much sweeter (and cheaper) fructose and high fructose corn syrup (HFCS).
Nowadays sugar is added to 75% of packaged foods and beverages in grocery stores.[1] Novel, man-made foods account for much of the incredible 120 lb/55kg of the sweet stuff many of us consume in a year (a 30-fold increase over 250 years[2]). From an evolutionary stance, it would be remarkable if a dietary change on this scale did not impact our health in some way.
In fact, a solid body of research now links rising consumption of added sugar to the epidemic of chronic, mostly preventable, diseases that most of us eventually die of (73% of global deaths in 2017)
Heart disease (No. 1 killer), obesity, type 2 diabetes, non-alcoholic fatty liver disease, Alzheimer’s, and certain cancers, have spread worldwide in parallel with the western diet of fast/processed foods, snacks and sugary drinks.
These ‘lifestyle’ diseases build up silently in our bodies over many years, only to reduce our healthy lifespan in mid-later life. Sadly, they are now appearing much earlier, in young adults and children.
Above all, since 1975, obesity has tripled. Today a staggering 40% of the world’s adults are classified as overweight, and one third of those as obese[3]. Moreover, a major increase in overweight children and adolescents suggests that rates will continue to climb sharply.
Becoming overweight from over-eating is commonly viewed as the No. 1 cause of chronic disease. The argument that a ‘calorie is a calorie’, blames us as consumers for our greed and lack of control.
However, there is mounting evidence that a high sugar intake is a health risk factor independent of excess weight and calorie intake. Many scientists believe that sugar, fructose in particular, disrupts metabolism by raising blood sugar, blood pressure, and cholesterol or triglyceride levels. As a result, this can trigger insulin resistance when cells in your muscles, body fat and liver start ignoring the signal from insulin to mop up glucose in the blood.
In turn, insulin resistance leads to the onset of many chronic diseases[4] including heart disease[5], diabetes[6], fatty liver disease[7], and dementia[8], even in people who are not overweight[9].
In other words, sugar can make you sick even if it doesn’t make you fat!
It’s easy to under-estimate how much sugar you are consuming. As well as the obvious; cakes, pastries, chocolates, sodas and fruit juices etc, added sugars are hidden in processed foods, even those labelled ‘healthy’ or ‘natural’ such as breakfast cereals, granola bars and yogurts.
Eating these can often push you over the six teaspoons of added sugar a day (5% of total calories) that most experts and the World Health Organisation recommend as an upper limit. If you get 10–25% of your daily calories from sugar, you run a 30% higher risk of dying of heart disease. Moreover, the risk triples if, like many people, you typically get more than 25% of your calories (about 20 teaspoons) from sugar. [10].
Chances are that having totted up your daily intake, you’ve realised that you regularly eat more than six teaspoons of added sugar a day. (Average consumption in the UK is 23 teaspoons; in the US 17-24 teaspoons, and is forecast to rise exponentially in the rest of the world as the price of sugar continues to fall).
Just as you want to avoid weight piling up inexorably over the years, so you want to prevent insulin resistance from building up silently. As we have seen insulin resistance is leading to a cascade of chronic health issues in increasingly younger populations.
If you do decide that the time has come to cut down, it may help to keep a food diary for a week or so and then settle on a date to start implementing some of these easy fixes:-
Let us have your favourite tips for tackling a sweet tooth in the comments below.
Check our latest healthier sweet eats under Recipes
[1]Ng SW, Slining MM, Popkin BM. Use of caloric and noncaloric sweeteners in US consumer packaged foods, 2005–2009. Journal of the Academy of Nutrition and Dietetics. 2012; 112:1828–34. e1–6. PubMed: 23102182
[2] Dinicolantonio, James & Lucan, Sean & O’Keefe, James. (2015). The Evidence for Saturated Fat and for Sugar Related to Coronary Heart Disease. Progress in Cardiovascular Diseases. 58. 10.1016/j.pcad.2015.11.006.
[3] https://www.statista.com/statistics/1065605/prevalence-overweight-people-worldwide-by-age/
[4] Mirtschink P, Jang C, Arany Z, Krek W. Fructose metabolism, cardiometabolic risk, and the epidemic of coronary artery disease. Eur Heart J. 2018;39(26):2497-2505. doi:10.1093/eurheartj/ehx518
[5] Yang Q, Zhang Z, Gregg EW, Flanders WD, Merritt R, Hu FB. Added sugar intake and cardiovascular diseases mortality among US adults. JAMA Intern Med. 2014;174(4):516-524. doi:10.1001/jamainternmed.2013.13563
[6] Imamura F, O’Connor L, Ye Z, et al. Consumption of sugar sweetened beverages, artificially sweetened beverages, and fruit juice and incidence of type 2 diabetes: systematic review, meta-analysis, and estimation of population attributable fraction. BMJ. 2015;351:h3576. Published 2015 Jul 21. doi:10.1136/bmj.h3576
[7] Jensen T, Abdelmalek MF, Sullivan S, et al. Fructose and sugar: A major mediator of non-alcoholic fatty liver disease. J Hepatol. 2018;68(5):1063-1075. doi:10.1016/j.jhep.2018.01.019
[8] Stephan BC, Wells JC, Brayne C, Albanese E, Siervo M. Increased fructose intake as a risk factor for dementia. J Gerontol A Biol Sci Med Sci. 2010;65(8):809-814. doi:10.1093/gerona/glq079
[9] Stanhope KL. Sugar consumption, metabolic disease and obesity: The state of the controversy. Crit Rev Clin Lab Sci. 2016;53(1):52-67. doi:10.3109/10408363.2015.1084990